The phytotoxic effect of herbicides applied post-emergence was investigated in a herbicide sensitivity experiment set up on parental maize genotypes in Martonvásár. A total of 48 Martonvásár inbred lines and 12 single line crosses were included in small-plot experiments set up in two replications. Ten herbicides were applied at the normal a...uthorised rate and at twice this quantity. Compounds intended for pre-emergence application were applied when maize was in the 3–4-leaf stage and post-emergence herbicides in the 7–8-leaf stage of development. The extent of phytotoxicity was scored two weeks after treatment. Some of the herbicides tested are not authorised for use in seed production fields, but it is important to know how the parental genotypes respond to all types of herbicides. Phytotoxic symptoms of varying intensity were only observed on a third of the 60 parental genotypes examined; the majority of the lines exhibited no reaction to any of the herbicides. Averaged over the 60 genotypes the level of phytotoxic damage was less than 10% for the single dose. When the double dose was applied somewhat more severe damage was induced by products containing Mesotrione + Nicosulfuron or Foramsulfuron + Isoxadifen-ethyl, but this was still below 15%. The herbicide dose had a three times stronger influence on the intensity of the symptoms than the type of herbicide. With the exception of Topramezone, there was a significant difference between the effects of the normal and double doses. The greatest dose effect differences, in decreasing order, were observed for Mesotrione + Nicosulfuron, Foramsulfuron + Isoxadifen-ethyl. Nicosulfuron and Mesotrione + Terbutylazine. The Mesotrione + Terbutylazine active ingredient combination only caused mild (<10%) symptoms on a total of 11 genotypes, while the Mesotrione + Nicosulfuron combination induced more severe phytotoxic symptoms on 26 lines. When Nicosulfuron was applied alone it caused milder symptoms on fewer genotypes than in combination with Mesotrione. Among compounds of the sulphonyl-urea type, the least severe symptoms on the fewest genotypes were recorded in the case of Prosulfuron.
The herbicide tolerance levels of 49 Martonvásár inbred parents were examined in Martonvásár in a herbicide susceptibility trial in 2016. The normal dosage recommended in the permit documentations and double dosage were used for the 12 small-plot herbicide treatments performed in two repetitions. Spraying of early post-emergent herbicides w...as carried out in the 1–2-leaf stage, while post-emergent treatments were applied in the 7–8-leaf stage of maize. The extent of phytotoxicity was scored for the early post-emergent herbicides two and four weeks after treatments and for the post-emergent herbicides two weeks after treatments, respectively. Some of the herbicides examined are not approved in seed production; however it is important to know the reaction of maize parent genotypes for every type of herbicides. The active agent topramezone was withdrawn from the market in 2015, but it was included in the trials as its usage was allowed until stocks run out in 2016. The herbicide agents were examined as follows: mesotrione + S-metolachlor + terbutylazine; isoxaflutol + tiencarbazon methyl + cyprosulfamide; isoxaflutol + cyprosulfamide; mesotrione + terbuthylazine; tembotrione + isoxidifen-ethyl; mesotrione + nicosulfuron; prosulfu ron; nicosulfuron +prosulfuron + dicamba; bentazone + dicamba; nicosulfuron; topramezone; foramsulfuron + isoxadifen-ethyl.
Among early post-emergent herbicides, isoxaflutol + cyprosulfamide caused the less phytotoxic damage in the genotypes. The large amount of precipitation during the spring facilitated the infiltration of the active ingredient S-metolachlor, used regularly and successfully also in seed production, into the root zone, resulting in phytotoxic symptoms on susceptible inbred lines at the time of the first inspection. These genotypes recovered by the end of the vegetation period. The spring weather was cooler than usual, retarding the development of maize and thus led to the slower fermentation of herbicide active ingredients, accordingly, all of the post-emergent herbicides caused visible phytotoxic symptoms on some of genotypes. The most severe damages were generally caused by the double dosage of nicosulfuron + prosulfuron + dicamba, nicosulfuron, and foramsulfuron + isoxadifen-ethyl.